"Molybdenum deficiency" from_date:2012

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                            1
                            2016Frontiers in neurology
                            Molybdenum Deficiency Produces Motor Nervous Effects That Are Consistent with Amyotrophic Lateral Sclerosis
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                            4
                            2018NPJ Parkinson's disease
                            Astrocyte dysfunction following molybdenum-associated purine loading could initiate Parkinson’s disease with dementia Sporadic or idiopathic Parkinson's disease is a movement disorder with a worldwide distribution, a long pre-clinical latent period and a frequent association with dementia. The combination of molybdenum deficiency and purine ingestion could explain the movement disorder , the distribution, the latent period and the dementia association. Recent studies in sheep have shown that molybdenum deficiency enables some dietary purines to accumulate in the central nervous system. This causes astrocyte dysfunction, altered neuromodulation and eventually irreversible central nervous system disease. Humans and sheep share the ability to salvage purines and this ability places humans at risk
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                            . Resting cells cultivated in a molybdenum-deficient medium had low nicotine transformation activity, and excess molybdenum was detected in the purified VppA by inductively coupled plasma-mass spectrometry analysis. Thus, it is demonstrated that VppA is a two-component molybdenum-containing hydroxylase.
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                            or molybdenum deficiency which may accompany this in children aged between 6 and 12 years in two schools in the province of Hatay (endemic goitre region). This study is a case-control field-study in which students aged between 6 and 12 years were included. One hundred fourteen subjects from the village of Tanışma related to the center of our province and 100 subjects from the city center of Hatay (Antakya
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                            IN THIS CHAPTER Overview of Minerals Chromium Deficiency Chromium Toxicity Copper Deficiency Copper Toxicity Wilson Disease Fluorine Deficiency Fluorine Toxicity Iodine Deficiency Iodine Toxicity Iron Deficiency Iron Toxicity Manganese Deficiency Manganese Toxicity Molybdenum Deficiency Molybdenum Toxicity Selenium Deficiency Selenium Toxicity Zinc Deficiency Zinc Toxicity Zinc Deficiency By Larry E. Johnson
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                            of Minerals Chromium Deficiency Chromium Toxicity Copper Deficiency Copper Toxicity Wilson Disease Fluorine Deficiency Fluorine Toxicity Iodine Deficiency Iodine Toxicity Iron Deficiency Iron Toxicity Manganese Deficiency Manganese Toxicity Molybdenum Deficiency Molybdenum Toxicity Selenium Deficiency Selenium Toxicity Zinc Deficiency Zinc Toxicity Wilson Disease (Wilson's Disease; Inherited Copper Toxicity
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                            / * * * * * OTHER TOPICS IN THIS CHAPTER Overview of Minerals Chromium Deficiency Chromium Toxicity Copper Deficiency Copper Toxicity Wilson Disease Fluorine Deficiency Fluorine Toxicity Iodine Deficiency Iodine Toxicity Iron Deficiency Iron Toxicity Manganese Deficiency Manganese Toxicity Molybdenum Deficiency Molybdenum Toxicity Selenium Deficiency Selenium Toxicity Zinc Deficiency Zinc Toxicity Overview
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                            2012Wikipedia
                            ] molybdenum deficiency / molybdenum toxicity[33] Selenium 0.055 0.4; 0.3 Essential to activity of antioxidant enzymes like glutathione peroxidase Brazil nuts, seafoods, organ meats, meats, grains, dairy products, eggs[34