"Pulmonary edema" from_date:2012

Nitrates for Acute Pulmonary Edema, Diuretic Prescriptions for CHF Prevention, POCUS for CHF Diagnosis Emergency Medicine Journal Club ARCHIVES/ CLINICAL TOOLS/ RESIDENTS/ PROCEDURES/ LLSA/ EPT  Nitrates for Acute Pulmonary Edema, Diuretic Prescriptions for CHF Prevention, POCUS for CHF DiagnosisThis month JC will be looking at another “bread and butter” ED complaint namely, ED management of the acute heart failure patient.  Kelly GS, et al., Low- versus high-dose nitroglycerin infusion in the management of acute pulmonary edema. Am J Emerg Med. 2023 Mar;65:71-75. AppraisalFaselis C, et al., Loop Diuretic Prescription and 30-Day Outcomes in Older Patients With Heart Failure. J Am Coll Cardiol. 2020 Aug 11;76(6):669-679. AppraisalMcGivery K et al., Emergency department ultrasound

Noninvasive Positive Pressure Ventilation for Cardiogenic Pulmonary Edema HomeReviewsTherapy (NNT) ReviewsDiagnosis (LR) ReviewsAboutThe NNT, ExplainedThe NNT Rating SystemtheNNT Editorial ProcessThe NNT Intervention QuizAbout theNNT TeamSubmit an articleAccountLoginSign upContactDONATENoninvasive Positive Pressure Ventilation for Cardiogenic Pulmonary EdemaReduces hospital mortality and endotracheal intubationsBenefits in NNT171 in 17 were helped (deaths prevented)131 in 13 were helped (endotracheal intubations prevented)Harms in NNTNo one was harmedView As: NNT %SourceBerbenetz N, Wang Y, Brown J, et al. Non-invasive positive pressure ventilation (CPAP or bilevel NPPV) for cardiogenic pulmonary oedema. Cochrane Database Syst Rev. 2019;4(4):CD005351.Study Population: 2664 adult participants

Sympathetic Crashing Acute Pulmonary Edema: High-Dose Vs. Low-Dose Nitro Sympathetic Crashing Acute Pulmonary Edema: High-Dose Vs. Low-Dose Nitro - REBEL EM - Emergency Medicine BlogSkip to content * Home * Blog * COVID-19 * Submit a Post * CAST * REVIEWS * PREZ * CRIT * CME/CEH * CORE * About * About Us * Meet The Team * Friends of REBEL EM * REBEL Swag * Disclaimer Menu * Home * Blog * COVID-19 * Submit a Post * CAST * REVIEWS * PREZ * CRIT * CME/CEH * CORE * About * About Us * Meet The Team * Friends of REBEL EM * REBEL Swag * Disclaimer * June 20, 2024 Sympathetic Crashing Acute Pulmonary Edema: High-Dose Vs. Low-Dose Nitro * Written byMarco Propersi * REBEL EM * Medical Category:Resuscitation, Thoracic and Respiratory Background: Sympathetic Crashing Acute Pulmonary Edema (SCAPE) results

Emergency medicine updates: Sympathetic crashing acute pulmonary edema. Patients with heart failure exacerbation can present in a variety of ways, including sympathetic crashing acute pulmonary edema (SCAPE). Emergency physicians play a key role in the diagnosis and management of this condition. This narrative review evaluates key evidence-based updates concerning the diagnosis and management of SCAPE for the emergency clinician. SCAPE is a subset of acute heart failure, defined as a patient with sudden, severe pulmonary edema and hypertension, resulting respiratory distress, and hypoxemia. This is associated with significantly elevated afterload with fluid maldistribution into the pulmonary system. Evaluation and resuscitation should occur concurrently. Laboratory assessment

Inflammatory indexes in emergency patients with hypertensive pulmonary Oedema: A critical insight. Heart failure (HF) is a prevalent and severe condition with high hospitalization and mortality rates, especially in developing countries. Inflammation plays a crucial role in its aetiology. Hypertensive pulmonary oedema, a severe form of acute decompensated heart failure (ADHF), lacks a definitive scoring system for predicting hospital admission outcomes. This study aims to evaluate the prognostic value of systemic inflammatory indexes (SII), systemic inflammation response index (SIRI), neutrophil/lymphocyte ratio (NLR), platelet/lymphocyte ratio (PLR), and multi-inflammatory indexes (MII-1, MII-2, MII-3) in patients with hypertensive pulmonary oedema. We conducted a retrospective observational

Negative pressure pulmonary edema resulting from upper airway obstruction during the post-anesthesia recovery period: a case series and literature review. Negative pressure pulmonary edema (NPPE) is a non-cardiogenic pulmonary edema primarily resulting from upper airway obstruction, particularly following laryngospasm during the post-anesthesia recovery period. We report five cases of NPPE that occurred during recovery from general anesthesia at our institution. Among these, four cases were attributed to glossoptosis following extubation, while one case resulted from laryngeal mask displacement. All patients exhibited typical clinical features and imaging findings consistent with pulmonary edema, and none experienced poor prognoses. Glossoptosis and improper positioning of the laryngeal mask

Postoperative pulmonary edema and pulmonary hemorrhage following hysteroscopy. Hysteroscopic surgery, a minimally invasive technique prevalent in gynecology, exhibits an overall complication rate between 0.22% and 3.7%. Pulmonary bleeding post-hysteroscopy represents an exceedingly rare complication with only three reported cases to date; two involving high-viscosity solutions and one . Diagnostic procedures included chest CT, echocardiography, and complete blood count. The chest CT illustrated diffusely reticular and patchy ground-glass opacities in both lungs, confirming the diagnosis of pulmonary edema with pulmonary bleeding. Treatment with dexamethasone and furosemide led to rapid improvement, meeting discharge criteria within 24 h post-surgery. Postoperative follow-ups showed

Respiratory rate-oxygenation index and National Early Warning Score 2 score are associated with orotracheal intubation in patients with cardiogenic pulmonary oedema. This study evaluates the association between the respiratory rate-oxygenation (ROX) index and the National Early Warning Score 2 (NEWS2) score with orotracheal intubation (OTI) and in-hospital mortality in patients with cardiogenic pulmonary oedema (CPE). This retrospective observational study enrolled patients aged 18 years or older who had developed CPE at admission or during hospital stay. Demographic, clinical, and laboratory data were collected within the first 24 h of CPE onset from the electronic records. The outcomes needed for OTI during 24 h after CPE diagnosis and in-hospital mortality were also collected. The ROX index

Morphine use in acute cardiogenic pulmonary oedema: is MIMO trial enough to change practice? Letter regarding the article 'Midazolam versus morphine in acute cardiogenic pulmonary oedema: results of a multicentre, open-label, randomized controlled trial'

High-flow nasal cannula oxygen versus noninvasive ventilation for the management of acute cardiogenic pulmonary edema: a randomized controlled pilot study. Whether high-flow nasal oxygen can improve clinical signs of acute respiratory failure in acute heart failure (AHF) is uncertain. To compare the effect of high-flow oxygen with noninvasive ventilation (NIV) on respiratory rate in patients

Evaluation of HACOR scale as a predictor of non-invasive ventilation failure in acute cardiogenic pulmonary oedema patients: A prospective observational study. Acute cardiogenic pulmonary oedema (ACPO) is a common indication for non-invasive ventilation (NIV) in the emergency department (ED). HACOR score of >5 is used to predict NIV failure. The predictive ability of HACOR may be affected by altered physiological parameters in ACPO patients due to medications or comorbidities. To validate the HACOR scale in predicting NIV failure among acute cardiogenic pulmonary oedema (ACPO) patients. This is a prospective, observational study of consecutive ACPO patients requiring NIV admitted to the ED. Primary outcome was the ability of the HACOR score to predict NIV failure. Clinical, physiological

Effect of furosemide in the treatment of high-altitude pulmonary edema. High-altitude pulmonary edema (HAPE) refers to the onset of breathlessness, cough, and fever at rest after arriving at high altitudes. It is a life-threatening illness caused by rapid ascent to high altitudes. Furosemide is controversial in HAPE treatment but is routinely used in China. Further research is needed to assess . Subgroup analysis showed that the longer the duration of furosemide use, the more pronounced the improvement in lung CT severity scores. But there were no significant differences in length of hospital stay and in-hospital mortality between the two groups. Furosemide helps alleviate pulmonary edema in HAPE patients, but further research is needed to clarify its impact on prognosis.

Calcium-activated potassium channels as amplifiers of TRPV4-mediated pulmonary edema formation in male mice. As a mechanosensitive cation channel and key regulator of vascular barrier function, endothelial transient receptor potential vanilloid-type 4 (TRPV4) contributes critically to ventilator-induced lung injury (VILI) and edema formation. Ca2+ influx via TRPV4 can activate Ca2+-activated K

Static Immersion and negative static lung load (SLL) induced right ventricle systolic function adaptation. A novel risk factor for Immersion Pulmonary Edema (IPE). Immersion pulmonary edema (IPE) is a form of hemodynamic edema, likely involves individual susceptibility. Can assessing right ventricular systolic adaptation during immersion be a marker for IPE susceptibility? Twenty-eight

Lung ultrasound diagnosis of pulmonary edema resulting from excessive fluid absorption during hysteroscopic myomectomy: a case report. Hysteroscopic surgery is a safe procedure used for diagnosing and treating intrauterine lesions, with a low rate of intraoperative complications. However, it is important to be cautious as fluid overload can still occur when performing any hysteroscopic surgical technique. In this case report, we present a unique instance where lung ultrasound was utilized to diagnose pulmonary edema in a patient following a hysteroscopic myomectomy procedure. The development of pulmonary edema was attributed to the excessive absorption of fluid during the surgical intervention. By employing lung ultrasound as a diagnostic tool, we were able to promptly identify and address

The predictive capacity of biomarkers for clinical pulmonary oedema in patients with severe falciparum malaria is low: a prospective observational study. Pulmonary oedema is a feared and difficult to predict complication of severe malaria that can emerge after start of antimalarial treatment. Proinflammatory mediators are thought to play a central role in its pathogenesis. An exploratory study was conducted to evaluate the predictive capacity of biomarkers for development of clinical pulmonary oedema in patients with severe falciparum malaria at two hospitals in Bangladesh. Plasma concentrations of interleukin-6 (IL-6), IL-8, tumour necrosis factor (TNF), soluble Receptor of Advanced Glycation End-products (sRAGE), surfactant protein-D (SP-D), club cell secretory protein (CC16), and Krebs von den

Severe postoperative negative pressure pulmonary edema: a case report. Postoperative negative pressure pulmonary edema (NPPE) can occur in any patient undergoing general anesthesia. There are several risk factors for it, especially postoperative laryngospasm. The disease is usually benign and quickly reversible. In our case the severity and need for advanced critical care therapy was unusual. We report a severe case of postoperative negative pressure pulmonary edema in a 62-year-old male patient undergoing elective right-sided retroperitoneoscopic adrenalectomy. The patient developed a severe case of acute respiratory distress syndrome (ARDS) after postoperative laryngospasm, possibly in conjunction with a suspected anaphylactic reaction. The patient was consequently treated with a combination

Retracted: Research Progress on the Mechanism of Right Heart-Related Pulmonary Edema. [This retracts the article DOI: 10.1155/2022/8947780.].

Midazolam versus morphine in acute cardiogenic pulmonary edema patients with and without atrial fibrillation: findings from the MIMO trial. The MIMO clinical trial showed that patients with acute cardiogenic pulmonary edema (ACPE) treated with midazolam had fewer serious adverse events than those treated with morphine. Atrial fibrillation (AF) is a common comorbidity in heart failure and affects

Taming the Tiger: Ultra high dose nitroglycerin in managing sympathetic crashing acute pulmonary edema patient. Sympathetic Crashing Acute Pulmonary Edema (SCAPE) lies on the end of the acute heart failure syndrome spectrum with pulmonary edema in all lung zones. NTG at lower doses (10-20 μg/min) cause preload reduction, and at higher doses (> 100 μg/min) causes after-load reduction by arterial is pathologically elevated afterload due to systemic vasoconstriction and hypertension. SCAPE patients may be euvolemic, hypovolemic or hypervolemic. The problem is shift of fluid into the lungs rather than hypervolemia. The emphasis on treating pulmonary edema has shifted from diuretics to vasodilators, especially high-dose nitrates, combined with non-invasive positive pressure ventilation. This is the first