"Salicylate poisoning" from_date:2012

Salicylate poisoning Salicylate poisoning - Symptoms, diagnosis and treatment | BMJ Best PracticeSkip to main contentSkip to search * English (US)EnglishPortuguês中文 * Log in * Personal account * Access through your institution(Open Athens) * Subscribe * Access through your institution * Log in * English (US)EnglishPortuguês中文 HomeSearchSearchHome * About usOverviewWhat is BMJ Best Practice?Our impactCustomer stories * Browse clinical contentRecent updatesSpecialtiesTry a free topicPatient informationVideosCalculators * What’s newClinical updatesNewsPodcast * AccessLog in via...Personal subscription or user profileAccess through your institutionAccess codeSubscribeFree trialHow do I get access?Download the app * HelpFAQsHow do I get access?Contact us Salicylate poisoning Menu Close * Overview

Severe Salicylate Poisoning Due to Teaberry Flavoring Ingestion: A Case Report. There are currently many commercial uses for methyl salicylate, including fragrances, flavoring, topical pain relief, and polymer restoration. Most recorded cases of human toxicity due to methyl salicylate are the result of topical or accidental exposures. However, this is a previously unreported case of an adult the single hemodialysis treatment with concomitant serum and urine alkalization, the patient had progressive improvement of salicylate concentrations. He was discharged on hospital day 3 in normal condition. WHY SHOULD AN EMERGENCY PHYSICIAN BE AWARE OF THIS?: Emergency physicians should be aware of novel etiologies of life-threatening salicylate poisoning.

Tracheal Intubation and Mechanical Ventilation in Adults with Severe Salicylate Poisoning. Salicylate poisoning may lead to critical acid-base disturbances. Tracheal intubation and mechanical ventilation for patients with severe salicylism has been strongly discouraged. This study aims to describe pH trends, complications, and outcomes in a cohort of salicylate-poisoned patients who were intubated. This retrospective observational study included adults presenting to the emergency department (ED) with severe salicylate poisoning (serum salicylate concentration >40 mg/dL and admission to an intensive care unit) over a 14-year period (2007-2021). The primary and secondary outcomes were the change in serum pH and the occurrence of severe complications (systolic blood pressure <80 mm Hg

Salicylate poisoning Salicylate poisoning - Symptoms, diagnosis and treatment | BMJ Best PracticeSkip to main contentSkip to search * About us * Help * Subscribe * Access through your institution * Log inBMJ Best Practice * Help * Getting started * FAQs * Contact us * Recent updates * Specialties * Calculators * Patient leaflets * Videos * Evidence * Drugs * Recent updates fatal. Patients can present acutely after accidental or intentional overdose. Chronic exposure to too much salicylate is an important, but difficult, diagnosis. Consider salicylate poisoning in any patient presenting with a history of unknown toxin ingestion or exposure, particularly in the presence of an unexplained acid-base disturbance. Consult the local poison control centre and/or toxicologist

Salicylate Poisoning We value your privacyWe and our partners store and/or access information on a device, such as cookies and process personal data, such as unique identifiers and standard information sent by a device for personalised ads and content, ad and content measurement, and audience insights, as well as to develop and improve products. With your permission we and our partners may use , hypokalaemia, hyponatraemia/hypernatraemia, hypocalcaemia, thrombocytopenia, abnormal blood coagulation (increased prothrombin ratio/INR), disseminated intravascular coagulation, acute kidney injury and non-cardiac pulmonary oedema.CNS: confusion, disorientation, coma and convulsions (less common in adults than in children).Chronic salicylate poisoning[2]Chronic salicylate poisoning is more common

The association of hemodialysis and survival in intubated salicylate-poisoned patients. Salicylate poisonings are common due to their multiple uses and wide availability. The variation of presenting symptoms contributes to inconsistent treatments in the emergency department. Patients with severe salicylate overdose require a high minute ventilation. Early in the course of an overdose, a patient

Salicylate Poisoning Potential of Topical Pain Relief Agents: From Age Old Remedies to Engineered Smart Patches The pain relief capabilities of methyl salicylate are well established and a multitude of over-the-counter products populate pharmacy shelves. Over-application of the topical preparation containing the drug, or its accidental ingestion, invariably result in salicylate poisoning

Hypercapnea and Acidemia despite Hyperventilation following Endotracheal Intubation in a Case of Unknown Severe Salicylate Poisoning Salicylates are common substances for deliberate self-harm. Acute salicylate toxicity is classically associated with an initial respiratory alkalosis, followed by an anion gap metabolic acidosis. The respiratory alkalosis is achieved through hyperventilation, driven by direct stimulation on the respiratory centers in the medulla and considered as a compensatory mechanism to avoid acidemia. However, in later stages of severe salicylate toxicity, patients become increasingly obtunded, with subsequent loss of airway reflexes, and therefore intubation may be necessary. Mechanical ventilation has been recommended against in acute salicylate poisoning, as it is believed

Acute Salicylate Poisoning: Risk Factors for Severe Outcome Salicylate poisoning remains a significant public health threat with more than 20,000 exposures reported annually in the United States. We aimed to establish early predictors of severe in-hospital outcomes in Emergency Department patients presenting with acute salicylate poisoning. This was a secondary data analysis of adult salicylate % sensitivity, 67% specificity). In adult Emergency Department patients with acute salicylate poisoning, independent predictors of severe outcome were older age and increased respiratory rate, as well as initial serum lactate, while initial salicylate concentration alone was not predictive.

diagnosis should be considered such as: lactic acidosis, inborn error of metabolism, alcoholic ketoacidosis, salicylate poisoning, or sepsis. Definition for DKA 1. Acidosis pH < 7.3 or HCO3 <15mmol/l 2. Ketones > 3.0 mmol/l 1.1 History • Polyuria • Polydipsia • Usual insulin regimen (if known diabetes) • Any triggers (e.g. Infections, stress) 1.2 Clinical signs • Acidotic (Kussmaul) breathing pattern

. There is insuffi cient clinical data to support or exclude the use of this therapy. The use of multiple-dose charcoal in salicylate poisoning is controversial (pg 75). Grade D, Level 4D Co-Administration of a cathartic to MDAC is unproven.Cathartics should not be administered to young children because of the propensity of laxatives to cause fl uid and electrolyte imbalance (pg 75 , decreased visual acuity or persistent irritation, refer to an ophthalmologist (pg 101). Grade D, Level 4Management of salicylate poisoningD Intubation of the salicylate-poisoned patient can be detrimental and should be avoided unless necessary (pg 101). Grade D, Level 3D If intubation and mechanical ventilation is necessary for severe obtundation, hypotension

Aspirin and Other Salicylate Poisoning Aspirin and Other Salicylate Poisoning - Injuries; Poisoning - MSD Manual Professional Edition MSD Manual Please confirm that you are a health care professionalYes No Leave this Site? The link you have selected will take you to a third-party website. We do not control or have responsibility for the content of any third-party site.Continue Cancel honeypot standard for trustworthy health information: verify here. Medical Topics & Chapters * A * B * C * D * E * F * G * H * I * J * K * L * M * N * O * P * Q * R * S * T * U * V * W * X * Y * Z 1. Professional / 2. Injuries; Poisoning / 3. Poisoning / 4. ... / 5. Aspirin and Other Salicylate Poisoning / * * * * * OTHER TOPICS

salicylate level is less than 30 mg/dL (2.2 mmol/L) and is falling at least 10% between assays in the absence of measures to enhance elimination. * Potassium stores are rapidly depleted in patients with salicylate intoxication and should be replaced with a goal serum level of 4.5 mEq/L. * Mechanical ventilation should be avoided if possible in cases of severe salicylate poisoning. Acidosis may rapidly glucose measurement, supplemental intravenous (IV) glucose (0.5-1 g/kg) should be administered. Refer to Box 144.2 in Rosen’s 9th edition for the detailed treatment of acute salicylate poisoning. A brief overview of the table is provided below. * Treat dehydration * Correct potassium depletion * Consider activated charcoal (AC) * Alkalinize urine * Initiate hemodialysis * Administer IV dextrose for any

deficiencyHyperventilatory States 1. Sepsis 2. Alcohol withdrawal 3. Salicylate poisoning 4. Neuroleptic malignant syndrome 5. Panic attacks 6. DKA 7. Hepatic comaHormonal and Endocrine Effects 1. Insulin loading 2. Glucose loading 3. Exogenous epinephrine 4. HyperparathyroidismMedicationsa 1. Diuretics 2. Chronic antacid ingestion 3. Steroids 4. Phosphate binders 5. Xanthine derivatives 6. Beta2-agonistsDisease States 1

* Ketoacidosis * Lactic Acidosis * Salicylate Poisoning * Methanol or Ethylene Glycol Poisoning * Show All * Medication * * * Medication Summary * Alkalinizing Agents * Carbonic Anhydrase Inhibitors * Antidiabetic Agents * Detoxification Agents * Show All * Questions & Answers * Tables * ReferencesOverview Practice EssentialsMetabolic acidosis is a clinical

Acetylsalicylic acid as a potential pediatric health hazard: legislative aspects concerning accidental intoxications in the European Union Acetylsalicylic acid is a frequently used medication worldwide. It is not used in pediatrics due its association with Reye syndrome. However, in case of pediatric intoxication, children are more fragile to salicylate poisoning because of their reduced ability

Salicylate Toxicity from Genital Exposure to a Methylsalicylate-Containing Rubefacient Methylsalicylate-containing rubefacients have been reported to cause salicylate poisoning after ingestion, topical application to abnormal skin, and inappropriate topical application to normal skin. Many over-the-counter products contain methylsalicylate. Topical salicylates rarely produce systemic toxicity when used appropriately; however, methylsaliclyate can be absorbed through intact skin. Scrotal skin can have up to 40-fold greater absorption compared to other dermal regions. We report a unique case of salicylate poisoning resulting from the use of a methylsalicylate-containing rubefacient to facilitate masturbation in a male teenager. Saliclyate toxicity has not previously been reported from

of unmeasured anions such as albumin or increased concentrations of unmeasured cations such as magnesium, calcium, or lithium. This teaching case describes a patient with aspirin (salicylate) poisoning whose anion gap was markedly reduced (-47 mEq/L). The discussion systematically reviews the possibilities and provides the explanation for this unusual laboratory result.

3. Low risk 1. Risperidone 8. Lithium 1. Organic brain syndrome 9. Zolpidem (Ambien) 1. Memory Impairment 2. Hallucinations IV. Medications: Analgesics 1. Salicylate Poisoning 2. NSAIDs (especially Indomethacin) 1. Delirium or confusion 2. Cognitive Impairment 3. Delirium 3. Opioid Analgesics (esp. Demerol, Fentanyl Patch) 1

Images * Extra: Related Studies * Extra: Navigation Tree advertisement II. Causes: Diaphoretic Skin (Mnemonic: SOAP) 1. Sympathomimetic Toxicity (contrast with Dry Skin with Anticholinergic Toxicity) 2. Organophosphate Poisoning 3. Aspirin (Salicylate Poisoning) 4. Phencyclidine (PCP) III. Causes: Needle Tracks 1. Intravenous Cocaine 2. Heroin 3. Dapsone IV. Causes: Red Skin 1