"Wernicke encephalopathy" from_date:2012

Wernicke's encephalopathy Wernicke's encephalopathy - Symptoms, diagnosis and treatment | BMJ Best PracticeSkip to main contentSkip to search * English (US)EnglishPortuguês中文 * Log in * Personal account * Access through your institution(Open Athens) * Subscribe * Access through your institution * Log in * English (US)EnglishPortuguês中文 HomeSearchSearchHome * About usOverviewWhat is BMJ Best encephalopathy. Eur J Neurol. 2010 Dec;17(12):1408-18. E, Farfar K, Errayes N, et al. Wernicke encephalopathy: an updated narrative review. Saudi J Med Med Sci. 2023 Jul-Sep;11(3):193-200. disorder is also frequently referred to as Wernicke-Korsakoff syndrome. Though Wernicke's encephalopathy and Korsakoff's psychosis (a condition characterised by amnesia and confabulation) are two clinically distinct

Wernicke's encephalopathy Wernicke's encephalopathy - Symptoms, diagnosis and treatment | BMJ Best PracticeSkip to main contentSkip to search * About us * Help * Subscribe * Access through your institution * Log inBMJ Best Practice * Help * Getting started * FAQs * Contact us * Recent updates * Specialties * Calculators * Patient leaflets * Videos * Evidence * Drugs * Recent PracticeTreatment algorithmACUTEmoderate or high suspicion of Wernicke's encephalopathylow suspicion of Wernicke's encephalopathy or mimics: at high risk for Wernicke's encephalopathyONGOINGpoor dietary intake and/or chronic alcoholism: at risk for Wernicke encephalopathyLog in or subscribe to access all of BMJ Best PracticeContributorsVIEW ALLAuthorsAbhay Moghekar, MB BSAssistant ProfessorDepartment

Wernicke Encephalopathy After Roux-en-Y Gastric Bypass Presenting with Altered Mental Status-A Video Case Report. Wernicke encephalopathy (WE) is a seldom encountered yet significant neuropsychiatric ailment resulting from a deficiency in thiamine (vitamin B1). While commonly linked with chronic alcoholism or insufficient dietary intake, instances of WE following bariatric and metabolic surgeries

Wernicke's encephalopathy with pinpoint pupils and diplopia. This case report presents the clinical findings of a female patient diagnosed with Wernicke's encephalopathy, characterized by pinpoint pupils. While pupillary changes can occur in Wernicke's encephalopathy, the presence of pinpoint pupils is exceedingly rare. In this report, we aim to document and discuss this unusual presentation

Wernicke Encephalopathy After Bariatric Surgery: a Literature Review. Wernicke encephalopathy (WE) is an acute neurological disorder classically characterized by ataxia, ophthalmoplegia, and altered mental status. This is caused by thiamine deficiency and is usually seen in malnourished populations. However, with the advent and rise of bariatric surgery in the last 50 years, WE has become

A Case of Sensorineural Hearing Loss in Wernicke Encephalopathy. Extremely rare cases of Wernicke encephalopathy (WE) can involve sensorineural hearing loss (SNHL). Here, we present a 46-year-old female with SNHL and clinical and radiologic suspicion for WE. After initiating thiamine therapy, the patient experienced robust improvement. Laryngoscope, 2023.

Hospital Outcomes in Medical Patients With Alcohol-Related and Non-Alcohol-Related Wernicke Encephalopathy. To conduct a nationwide retrospective cohort study to assess trends and hospitalization-associated outcomes in patients with Wernicke encephalopathy. In this nationwide retrospective cohort study, we used in-hospital claims data of patients hospitalized with Wernicke encephalopathy in Switzerland from January 1, 2012, to December 31, 2020. We estimated incidence rates per 100,000 person-years among the overall Swiss population stratified by alcohol and non-alcohol-induced Wernicke encephalopathy. The primary outcome was all-cause in-hospital mortality. Secondary outcomes included progression to Korsakoff syndrome and 1-year hospital readmission. We estimated odds ratios (ORs) for binary

What is the optimum thiamine dose to treat or prevent Wernicke's encephalopathy or Wernicke-Korsakoff syndrome? Results of a randomized controlled trial. The primary cause of Wernicke-Korsakoff syndrome (WKS) is thiamine deficiency, and more than 90% of cases are reported in alcohol-dependent patients. While observational studies show parenteral thiamine administration drastically reduced WKS -related mortality, relevant treatment trials have never been conducted to determine the optimum thiamine dose. Two double-blind, parallel groups, randomized controlled trials (RCTs) were conducted to determine the optimal thiamine dose required for (1) the prevention of Wernicke's encephalopathy (WE), the acute phase of WKS, in asymptomatic but "at-risk" alcohol misuse patients (Study 1) and (2

Bromisoval-induced bromism with status epilepticus mimicking Wernicke's encephalopathy: report of two cases. Bromine compounds are used in several drugs, including over-the-counter drugs. They sometimes cause intoxication known as bromism. Although the acute neurological symptoms and sequelae of bromism vary, few reports have mentioned acute encephalopathy. We report two cases of bromisoval -induced bromism with status epilepticus. Presence of pseudohyperchloremia and history of over-the-counter medication use guided the diagnosis. In the acute phase, our patients showed bilateral medial thalamic lesions on magnetic resonance imaging. The imaging findings were similar to those of Wernicke's encephalopathy. Although these findings improved in the chronic phase, neuropsychiatric sequelae

Wernicke's encephalopathy in women with hyperemesis gravidarum - Case series and literature review. Whilst nausea affects around 80% of pregnant women, hyperemesis gravidarum, an extreme form of the same, affects only 0.5% to 3%, but may lead to severe nutritional deficiency. Wernicke's encephalopathy is an acute neuropsychiatric disorder which occurs due to thiamine deficiency and needs emergency treatment to prevent neurological morbidity and mortality. Wernicke's encephalopathy is characterised by a clinical triad of oculomotor abnormalities, cerebellar dysfunction and altered mental state. Korsakoff's psychosis is a chronic condition and consequence of Wernicke's encephalopathy, resulting from its delayed treatment. Wernicke's encephalopathy is a well-known complication of chronic

Wernicke's encephalopathy in advanced cancer. This 54-year-old woman with metastatic breast cancer presented with severe confusion, incontinence and self-neglect. MRI suggested Wernicke's encephalopathy (WE) without any history of alcohol misuse. The patient's symptoms seemed to not respond to acute treatment. Cancer progression was assumed to be causing her condition and she was discharged

Wernicke Encephalopathy After Sleeve Gastrectomy. Wernicke encephalopathy (WE) is a rare and serious complication of obesity surgery. This is a report of one case of WE after a sleeve gastrectomy. The diagnosis is primarily clinical; the persistent and prolonged vomiting, and noncompliance to vitamin intake are two major risk factors to develop WE. It is especially important to rule out

Cognitive impairment severity in relation to signs of subclinical Wernicke's encephalopathy in HIV and alcoholism comorbidity. The comorbidity of HIV infection and alcoholism (ALC) is prevalent. Wernicke's encephalopathy, a neurological disorder resulting from thiamine depletion, has been generally associated with alcoholism but has also been reported in HIV infection. This study examined whether subclinical Wernicke's encephalopathy signs could contribute to the heterogeneity of cognitive and motor deficits observed in individuals with both disease conditions (HIV+ALC). Sixty-one HIV+ALC individuals and 59 controls were assessed on attention and working memory, production, immediate and delayed episodic memory, visuospatial abilities, and upper limb motor function. Using Caine criteria

Stability of high-dose thiamine in parenteral nutrition for treatment of patients with Wernicke's encephalopathy. Wernicke's encephalopathy is associated mainly with malnourishment in alcohol-dependent patients but can be caused also by cancer, Crohn's disease, gastrointestinal surgery or prolonged parenteral nutrition (PN) without adequate supplementation of vitamins. The disorder a high dose of thiamine (800 mg per bag, 320 mg/L) to PN admixtures in order to treat Wernicke's encephalopathy in malnourished patients. The study aimed to assess the stability of the physical properties of PN admixtures (pH, zeta potential, particle size) and to determine thiamine content using an HPLC method. Thiamine was found to degrade regardless of the PN admixture composition and storage

Wernicke's encephalopathy due to malnutrition and parenteral nutrition in a patient with cerebral infarction: A case report. Wernicke's encephalopathy (WE) is a severe neuropsychiatric disorder, which results from a nutritional deficiency of thiamine. The occurrence of WE is rarely reported in patients with cerebral infarction, who often have complications of malnutrition. Cerebral infarction

Wernicke encephalopathy associated with hyperemesis gravidarum. Emergency physicians frequently treat hyperemesis gravidarum and should be aware of possible complications. Wernicke encephalopathy secondary to thiamine deficiency should be considered in the differential diagnosis of acute encephalopathy in pregnant women. A seventeen-week pregnant 27-year-old woman presented to the Emergency -weighted signal in the central pons and medial thalami. Radiographic differential included central pontine myelinolysis, dysmyelinating conditions from malnutrition, toxic encephalopathy, and Wernicke encephalopathy. Thiamine level was below the limits of detection. Alcohol and urine drug screen were negative. Diagnosis was thiamine deficiency secondary to hyperemesis gravidarum with Wernicke

Wernicke Encephalopathy-Clinical Pearls. Wernicke encephalopathy (WE) was first described by Carl Wernicke in 1881. WE is caused by thiamine deficiency. Alcoholism is the most common etiologic factor associated with WE in the United States, but it can occur in any patient with a nutritional deficiency state such as hyperemesis gravidarum, intestinal obstruction, and malignancy. WE is a clinical

Hypothalamic sydrome as an initial presentation of Wernicke encephalopathy: A case report. Wernicke encephalopathy (WE) is a syndrome characterized by an acute or subacute onset of ataxia, ophthalmoplegia, and mental status changes. To our knowledge, hypothalamic syndrome is rare in WE. A 73-year-old female patient with acute cerebral infarct, who showed initial symptoms of vomiting, nausea

Wernicke encephalopathy concurrent with polyradiculoneuropathy in a young man after bariatric surgery: A case report. Bariatric surgery is the recommended treatment for morbid obesity because of its rapid and sustained body weight loss effect. Nutrient deficiency-related neurological complications after bariatric surgery are often disabling. Thus, early recognition of these complications is important. Neurological complications involving the central and peripheral nerve system after bariatric surgery were reported. However, the report on the clinical course of the concurrent involvement of central and peripheral nervous system is limited. We present a rare case of a patient who developed Wernicke encephalopathy concurrent with polyradiculoneuropathy after receiving bariatric surgery. A 22

Wernicke's Encephalopathy : MRI Wernicke's Encephalopathy : MRI - Sumer's Radiology Blog * Home * About Us * Sitemap * * * * * * Top Ad unit 728 × 90 * Neuroradiology * Chest Radiology * Musculoskeletal Radiology * GI Radiology * Genitourinary RadiologyRadiology Newsradiology HomeUnlabelledWernicke's Encephalopathy : MRIWernicke's Encephalopathy : MRITuesday, September 29, 2020This is an MRI image obtain in alcoholic beggar presented with ataxia, ophthalmoplegia and acute confusional state. Red arrow shows hyperintense signal around mamillary bodies and yellow arrow around periaqueductal area.Important Point :MRI in wernicke's encephalopathy shows hyperintense T2/FLAIR signal involving the mamillary bodies, dorsomedial thalami, tectal plate, periaqueductal area, and around 3rd