"Accessory pancreas"

Accessory pancreatic lobe in association with a gastric duplication cyst. Gastric duplication cysts are an extremely rare anomaly with few reported cases in association with accessory pancreatic tissue. Diagnosis can be challenging given a presentation of recurrent pancreatitis and resemblance to pancreatic pseudocysts. We report the case of a 6-year old boy with multiple episodes of pancreatitis who was discovered to have an accessory pancreatic lobe connected to a gastric duplication cyst, successfully treated with surgical excision.

Accessory Pancreatic Duct Patterns and Their Clinical Implications Accessory pancreatic duct (APD) designed to reduce the pressure of major pancreatic duct by forming a secondary drainage channel. Few studies have mentioned the variant types of accessory ducts and their mode of formation, some of these have a clear clinical significance. Present study is aimed to evaluate the possible variations in the APD and its terminations. Forty formalin fixed adult human pancreas with duodenum in situ specimens were studied by injecting 1% aqueous eosin, followed by piece meal dissection of the head of the pancreas from posterior surface. Formation, tributaries, relations, and the termination of the accessory pancreatic duct were noted and photographed. Accessory ducts revealed 50% belonged to long type

as the ampulla of Vater were carefully preserved. The proximal side of the main and accessory pancreatic duct was identified and sutured. Pancreatic anastomosis was performed using the technique of Bing's anastomosis.7 Fig. 1 Enhanced computed tomography (CT) scan showing atrophy of the pancreas, dilatation of the main pancreatic duct, and heterogeneous enhancement of the pancreatic head parenchyma Fig. 2

, including induction by primary acinar cell injury as a result of viral infections, drugs, ischemia, and direct trauma, as well as disruption of the ductal system and subsequent excretion of digestive enzymes from the acinar cells of the pancreas.Normally, the acinar cells release inactive enzymes into collecting ducts, which then drain into the main or accessory pancreatic ducts emptying directly

. Fluorodeoxyglucose-positron emission tomography/CT revealed an abnormal uptake of fluorodeoxyglucose, corresponding to the mass lesions. Upper gastrointestinal endoscopy revealed rough mucosa near the opening of the accessory pancreatic duct, and the mucosa biopsy exhibited adenocarcinoma with no mucin observed. The preoperative diagnosis was pancreatic intraductal tubulopapillary neoplasm (ITPN) with cancerous

for endoscopic treatment of CP. Given that a multitude of CP patients are accompanied by main pancreatic duct stricture and distortion, certain patients encounter failure in main papilla angiography or are unable to achieve deep cannulation of the pancreatic duct, thus necessitating endoscopic retrograde accessory pancreatic ductography via the accessory papilla to augment the success rate of endoscopic drainage. Presently, there lacks a large - scale case report concerning the proportion of patients receiving main and accessory pancreatic ductography treatment and the safety and efficacy of endoscopic retrograde accessory pancreatic ductography in the treatment of CP. This study endeavors to establish a prospective cohort to document the proportion of CP patients undergoing main and accessory

Pancreas divisum: correlation between anatomical abnormalities and bile precipitation in the gallbladder in seven patients Pancreas divisum is a genetic defect associated with recurrent acute pancreatitis due to insufficient drainage of the accessory pancreatic duct. Seven young patients diagnosed with pancreatic divisum and thickening of the gallbladder bile as shown on magnetic resonance

Adenocarcinoma of the minor duodenal papilla and its precursor lesions: a clinical and pathologic study. The minor duodenal papilla drains the accessory pancreatic duct of Santorini and lies proximal to the ampulla of Vater. Adenocarcinoma and its precursor lesions arising in the minor papilla are rare. Literature data thus far are limited to a few individual case reports, and the condition

with other anomalies, such as patent urachus, Meckel's diverticulum, mesenteric cyst, and accessory pancreas. Clinical manifestation of gastrointestinal duplication cysts was variable, and some of them were detected accidently. Thin- or thick-walled cystic structures adjacent to the wall of neighboring gastrointestinal segment were documented on diagnostic imaging. Ultrasound and computed tomography

). Contrast-enhanced axial CT scans through the pancreas show a 5.5-cm cystic tumor in the pancreatic head. Note the upstream, gross dilatation of the pancreatic duct. The accessory pancreatic duct is also dilated. View Media Gallery Pancreatic intraductal papillary mucinous tumor (IPMT). (Top) Superior mesenteric angiogram shows capillary vascularity in the mass in the pancreatic head during the arterial .)Pancreatic intraductal papillary mucinous tumor (IPMT). Contrast-enhanced axial CT scans through the pancreas show a 5.5-cm cystic tumor in the pancreatic head. Note the upstream, gross dilatation of the pancreatic duct. The accessory pancreatic duct is also dilated. View Media Gallery Pancreatic intraductal papillary mucinous tumor (IPMT). Contrast-enhanced axial CT scans through the pancreas show

release inactive enzymes into collecting ducts, which then drain into the main or accessory pancreatic ducts emptying directly into the duodenal lumen. If obstruction or disruption of these ducts occurs, the pancreatic secretions are activated within the parenchyma of the pancreas and initiate autodigestion of the pancreatic tissue.Interstitial edema is an early finding. Exacerbation of pancreatitis may

release inactive enzymes into collecting ducts, which then drain into the main or accessory pancreatic ducts emptying directly into the duodenal lumen. If obstruction or disruption of these ducts occurs, the pancreatic secretions are activated within the parenchyma of the pancreas and initiate autodigestion of the pancreatic tissue.Interstitial edema is an early finding. Exacerbation of pancreatitis may

portion of the duodenum; abnormal enhancement of the second portion of the duodenum; and cystic focus in the expected region of the accessory pancreatic duct. Strict criteria for diagnosis of PDP included presence of all three imaging features. Any case that did not fulfill the criteria was classified as CA. Sensitivity, specificity, positive predictive value, and negative predictive value

, and procedure duration. There was no significant difference between the groups with regard to visualization of the bile duct and/or the main and accessory pancreatic ducts, presence of pancreatic acinarization, number of injections into the pancreatic duct, total volume of contrast used, and procedure duration. Overall, post-ERCP pancreatitis occurred in 4 of the 370 patients (1.1%). The incidence

Pancreas * Annular pancreas * Accessory pancreas * Johanson–Blizzard syndrome * Rectovestibular fistula * Persistent cloaca * Rectal atresia Accessory

to the decision to perform an open exploration for suspicion of a pyloric duplication. An incision of the pyloric muscle exposed and confirmed a duplication cyst. Further exposure revealed accessory pancreatic tissue communicating from the cyst to the main body of the pancreas. The duplication cyst and accessory pancreas were resected, and the patient had an uneventful recovery. Pyloric duplication is a rare

III. The Surgical Significance of the Accessory Pancreas

ACCESSORY PANCREAS WITH INTUSSUSCEPTION

A STUDY OF THE ACCESSORY PANCREAS: WITH REPORT OF ONE CAUSING CONGENITAL PYLORIC STENOSIS

Persistent Omphalomesenteric with Accessory Pancreas