Cardiovascular Morbidity and Mortality After AmaurosisFugax. This cohort study examines the risk of cardiovascular morbidity and mortality in patients with amaurosisfugax compared with a matched control group.
Antiphosphospholipid syndrome presenting with amaurosisfugax and cotton wool spots To describe the importance of considering vaso-occlusive disease on the differential diagnosis of a patient presenting with amaurosisfugax (AF) and unilateral cotton wool spots (CWS). A 69-year-old female with history of obesity, hyperlipidemia and recent orthopedic surgery, presented with 3 days of worsening
Amaurosisfugax associated with congenital vascular defect A 68-year-old female with no significant past medical history presented with loss of vision in the lower half of her left eye that lasted <5 minutes. No abnormalities were found on ocular or physical exam. Computed tomography angiography and carotid ultrasound were performed, which confirmed the diagnosis as amaurosisfugax with two of an atherosclerotic plaque at the origin of the common carotid artery. This is an abnormal location for a plaque leading to amaurosisfugax compared to the most common location at the carotid bifurcation. Endarterectomy was not performed because of the difficult location of the plaque and tortuosity of the vessel. Rather, medical intervention with antiplatelet and lipid-lowering therapy was initiated to lower
Amaurosisfugax due to pleomorphic sarcoma in the left atrium This report describes a case of amaurosisfugax due to a rare primary cardiac sarcoma. A patient who was recently diagnosed with left atrial pleomorphic sarcoma presented with a chief complaint of multiple episodes of intermittent vision loss in the right eye during the course of radiation therapy. The authors postulate emboli from
Amaurosisfugax: risk factors and prevalence of significant carotid stenosis The purpose of this study was to describe clinical characteristics and prevalence of carotid stenosis in patients with amaurosisfugax (AF). Patients diagnosed with AF and subjected to carotid ultrasound in 2004-2010 in Sahlgrenska University Hospital, Gothenburg (n=302), were included, and data were retrospectively
Amaurosisfugax – delay between symptoms and surgery by specialty To describe the time course of management of patients with amaurosisfugax and analyze differences in management by different specialties. Patients diagnosed with amaurosisfugax and subjected to carotid ultrasound in 2004-2010 at the Sahlgrenska University Hospital, Gothenburg, Sweden (n=302) were included in this retrospective
AMAUROSISFUGAX IN OCULAR VASCULAR OCCLUSIVE DISORDERS: Prevalence and Pathogeneses. To investigate systematically the prevalence of amaurosisfugax (AF) in various ocular vascular occlusive disorders individually and to discuss the pathogeneses of each. The study comprised patients with central retinal artery occlusion (271 eyes), branch retinal artery occlusion (169 eyes), ocular ischemic . In giant cell arteritis, 32.4% of patients with ocular involvement had a history of AF or 26.5% of the involved eyes. Amaurosisfugax in central retinal artery occlusion, branch retinal artery occlusion, and nonarteritic anterior ischemic optic neuropathy is mostly because of transient embolism. The pathogenesis of AF in each ocular vascular occlusive disorder is discussed. Prevalence and pathogenesis
Recurrent AmaurosisFugax in a Patient after Stanford Type A Dissection Depending on Blood Pressure and Haemoglobin Level Purpose. A transient painless monocular visual loss due to a decrease in retinal circulation-also known as "amaurosisfugax"-often precedes acute territorial cerebral ischaemia. The case we present underlines the importance of a comprehensive diagnostic workup in patients with amaurosisfugax. Case Report. A 44-year-old man who had suffered from a dissection of the ascending aorta (Stanford Type A) five months ago presented with recurrent monocular vision problems. Episodes with sectional vision loss mainly occurred in combination with low blood pressure levels. Furthermore, the haemoglobin level was chronically low (Hb 9.7 mg/dL), and the patient was by mistake
of plaques may rupture and embolise to occlude intracranial arteries, causing a transient ischaemic attack or stroke, or occlude retinal arteries to cause transient monocular blindness (amaurosisfugax) or retinal strokes.History and examKey diagnostic factorsasymptomaticcervical bruitfocal neurological deficit lasting >24 hours (i.e., stroke)focal neurological deficit lasting <24 hours (i.e., transient
diagnostic factorssudden onset and brief duration of symptoms (minutes)patient/witness report of focal neurological deficitFull detailsOther diagnostic factorsunilateral weakness or paralysisdysphasiaataxia, vertigo, or loss of balancesudden transient loss of vision in one eye (amaurosisfugax)Full detailsRisk factorsatrial fibrillationvalvular diseasecarotid stenosisintracranial stenosisFull detailsLog
(TIA), including ocular or retinal stroke and amaurosisfugax; - subarachnoid haemorrhage (SAH): immediate management required at an admitting hospital. [2016] The guideline does not cover: - primary prevention of stroke; - detailed recommendations on (neuro-)surgical techniques (but the role of surgery is addressed); - management of children with stroke; - surgical or neuroradiological
symptomatic when correlated with findings like focal neurologic symptoms produced by ipsilateral transient ischaemic attacks, amaurosisfugax, or an ischaemic stroke in the carotid artery territory during the preceding six months. According to the NASCET criteria (North American Symptomatic Carotid Endarterectomy Trial [4]), a high-grade stenosis is characterised as a ≥ 70% stenosis (equivalent to a ≥ 85