Disseminated anetoderma in a patient with nodal Epstein-Barr virus-associated classical Hodgkin lymphoma: Anetodermic form of a concurrent discordant cutaneous marginal zone lymphoma. Patients with a lymphoma have an increased risk of developing a second lymphoproliferative disorder. The association of nodal Hodgkin lymphoma and primary cutaneous marginal zone lymphoma (MALT type) is exceptional , and only very few cases have been documented. Anetoderma represents a circumscribed loss or rarefication of elastic fibers. Different underlying processes may result in anetoderma, including cutaneous marginal zone lymphoma. We report a 50-year-old male patient with Epstein-Barr virus (EBV)-associated nodal Hodgkin lymphoma who presented with disseminated anetodermic skin lesions. Biopsies of the skin
A Clinicoimmunohistopathologic Study of Anetoderma: Is Protruding Type More Advanced in Stage Than Indented Type? . The clinical and histopathologic classification of anetoderma are not well characterized. . We aimed to investigate the clinical and histopathologic characteristics of anetoderma and to correlate clinical phenotypes with immunohistopathologic findings. . We retrospectively reviewed the medical records of 30 patients with anetoderma and performed immunohistochemistry for elastin, fibrillin-1, metalloproteinase- (MMP-) 2, MMP-7, MMP-9, and MMP-12, and tissue inhibitor of metalloproteinase- (TIMP-) 1 and TIMP-2. . Protruding type ( = 17) had a longer disease duration and more severe loss of elastin, without changes in fibrillin, than indented type ( = 13). MMP-2 and MMP-9 showed
%) were superficial hemangiomas, whereas 11 (19.0%) were combined-type. The median maximum diameter was 10.0 mm. Forty-five lesions (77.6%) exhibited various residual skin changes after PDL treatment, including anetoderma (53.5%), telangiectasia and erythema (43.1%), hyperpigmentation (34.5%), redundant skin (3.4%), and fibrofatty tissue (3.4%). Of these, the incidence of anetoderma and fibrofatty
that ACA is not the only possible presentation of LCLB. The diagnosis of ACA is often clinically missed for months or years, and may be mistaken at the inflammation phase for vascular disorders, erysipelas or bursitis/arthritis, and at the atrophic phase for lichen sclerosus atrophicus, morphoea or anetoderma. To our knowledge, no such tumorous LCLB has previously been described.
-2013 and were followed prospectively. The sample included seven patients (five male, two female) of mean age 38 years at diagnosis; two were aged <18 years. The lesion presented as a solitary patch/plaque with follicular accentuation in five patients, an infiltrated plaque devoid of hair in one and with follicular nodules in one. Four patients had alopecia, and one, secondary anetoderma. The lesion
intervention, juvenile xanthogranulomas (JXGs) flatten with time. Both cutaneous and extracutaneous lesions involute spontaneously within 3-6 years.Hyperpigmentation, mild atrophy, or anetoderma may persist.Lesions can recur after resection. The relapse rate is approximately 7%.In the absence of neurofibromatosis, no systemic health implications are involved, with a few rare exceptions.Vigilantly screen . This website also contains material copyrighted by 3rd parties.Close encoded search term (Dermatologic Manifestations of Juvenile Xanthogranuloma) and Dermatologic Manifestations of Juvenile Xanthogranuloma What to Read Next on Medscape Related Conditions and Diseases * Dermatologic Manifestations of Juvenile Xanthogranuloma * Juvenile Xanthogranuloma * Histiocytosis * Mastocytosis * Anetoderma
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parties.Close encoded search term (Dermatologic Manifestations of Juvenile Xanthogranuloma) and Dermatologic Manifestations of Juvenile Xanthogranuloma What to Read Next on Medscape Related Conditions and Diseases * Dermatologic Manifestations of Juvenile Xanthogranuloma * Juvenile Xanthogranuloma * Histiocytosis * Mastocytosis * Anetoderma * Dermatofibroma * Neonatal and Pediatric Lupus