"Hollenhorst plaque"

Teaching Video NeuroImages: Hollenhorst plaque.

Is Carotid Ultrasound Necessary in the Clinical Evaluation of the Asymptomatic Hollenhorst Plaque? (An American Ophthalmological Society Thesis) To evaluate the utility of carotid ultrasound in patients with asymptomatic Hollenhorst plaques. Retrospective chart review of 237 patients diagnosed with Hollenhorst plaques between 1996 and 2004. The baseline cardiovascular risk profile, medications was obtained from 32 symptomatic patients (39.6 ± 22.9 months) and 100 asymptomatic patients (41.3 ± 21.8 months). Vascular and neurologic event rates were similar between the two groups. Hollenhorst plaques are a marker of significant carotid disease irrespective of retinal symptoms. Carotid auscultation remains important in the examination of patients with Hollenhorst plaques and increases the yield

* Diagnosis of suspected carotid stenosis * Hollenhorst plaques (cholesterol emboli) or retinal neovascularity on retinal examination * Management of known carotid stenosis * Worsening neurologic symptoms or signs attributable to the anterior circulation * Initial baseline evaluation, and one additional evaluation during the first year following carotid revascularization * Surveillance of established

on nonvascular imaging • Diagnosis of suspected carotid stenosis o Hollenhorst plaques (cholesterol emboli) or retinal neovascularity on retinal examination • Management of known carotid stenosis o Worsening neurologic symptoms or signs attributable to the anterior circulation o Initial baseline evaluation and at 6 months following carotid revascularization • Surveillance of established carotid disease o

of all-cause and specific-cause mortality among U.S. adults. Individuals aged ≥ 40 years were included from the U.S. National Health and Nutrition Examination Survey, 2005-2008. RMA and its subtypes, including retinopathy, arteriovenous nicking (AVN), focal arteriolar narrowing (FAN) and Hollenhorst plaque (HP), were manually graded from retinal photographs. Associations between RMA and the risk of all

○Paralysis or paresthesias = Limb threatening ischemia Fundoscopic exam may show Roth’s spots (IE) or Hollenhorst plaques (atheromatous

myeloma * * Signs of diabetes mellitus * * Signs of hypertension * * Atheroemboli: Retinopathy (ie, Hollenhorst plaque in cholesterol microembolism) EarExamination of the patient’s ears may reveal the following signs: * * Hearing loss: Alport disease and aminoglycoside toxicity * * Mucosal or cartilaginous ulcerations: granulomatosis with polyangiitis (Wegener granulomatosis

to be present: a recent history of high-risk endovascular procedures, pathognomonic ischemic manifestations of extremities such as blue toes syndrome or livedo reticularis and Hollenhorst plaques on fundoscopy.The exclusion criteria were the presence of other treatment for ARD, such as LDL-apheresis sessions or cyclophosphamide and the lack of detailed follow-up, i.e., the lack of reporting of a key clinical

vessels 1. Retinal vessels with interrupted columns of blood appear as train box cars 2. Blood cells separate from serum 5. Hollenhorst Plaques (white punctate Cholesterol emboli) 1. "Glistening orange yellow flakes" 2. Represent fragmented emboli at arteriole bifurcations 4. Neck Exam 1. Carotid Bruit IX. Exam 1. See Eye Vital

-Retinal membrane 4. Retinal Artery Occlusion 1. Retina appears pale-gray due to Retinal edema 2. Central Macula (fovea) with cherry-red spot on white-yellow background 3. Constricted arterioles 4. Box-Carring of Retinal vessels 1. Retinal vessels with interrupted columns of blood appear as train box cars 5. Hollenhorst Plaques (emboli) 1. "Glistening orange

* * Signs of hypertension * * Atheroemboli: Retinopathy (ie, Hollenhorst plaque in cholesterol microembolism) EarsExamination of the patient’s ears may reveal the following signs: * * Hearing loss: Alport disease and aminoglycoside toxicity * * Mucosal or cartilaginous ulcerations: Granulomatosis with polyangiitis ( Wegener granulomatosis) Cardiovascular systemCardiovascular examination may reveal

* * Signs of hypertension * * Atheroemboli: Retinopathy (ie, Hollenhorst plaque in cholesterol microembolism) EarsExamination of the patient’s ears may reveal the following signs: * * Hearing loss: Alport disease and aminoglycoside toxicity * * Mucosal or cartilaginous ulcerations: Granulomatosis with polyangiitis ( Wegener granulomatosis) Cardiovascular systemCardiovascular examination may reveal

myeloma * * Signs of diabetes mellitus * * Signs of hypertension * * Atheroemboli: Retinopathy (ie, Hollenhorst plaque in cholesterol microembolism) EarExamination of the patient’s ears may reveal the following signs: * * Hearing loss: Alport disease and aminoglycoside toxicity * * Mucosal or cartilaginous ulcerations: granulomatosis with polyangiitis (Wegener granulomatosis

* * Signs of hypertension * * Atheroemboli: Retinopathy (ie, Hollenhorst plaque in cholesterol microembolism) EarsExamination of the patient’s ears may reveal the following signs: * * Hearing loss: Alport disease and aminoglycoside toxicity * * Mucosal or cartilaginous ulcerations: Granulomatosis with polyangiitis ( Wegener granulomatosis) Cardiovascular systemCardiovascular examination may reveal

myeloma * * Signs of diabetes mellitus * * Signs of hypertension * * Atheroemboli: Retinopathy (ie, Hollenhorst plaque in cholesterol microembolism) EarExamination of the patient’s ears may reveal the following signs: * * Hearing loss: Alport disease and aminoglycoside toxicity * * Mucosal or cartilaginous ulcerations: granulomatosis with polyangiitis (Wegener granulomatosis

* * Signs of hypertension * * Atheroemboli: Retinopathy (ie, Hollenhorst plaque in cholesterol microembolism) EarsExamination of the patient’s ears may reveal the following signs: * * Hearing loss: Alport disease and aminoglycoside toxicity * * Mucosal or cartilaginous ulcerations: Granulomatosis with polyangiitis ( Wegener granulomatosis) Cardiovascular systemCardiovascular examination may reveal

vessels 1. Retinal vessels with interrupted columns of blood appear as train box cars 2. Blood cells separate from serum 5. Hollenhorst Plaques (white punctate Cholesterol emboli) 1. "Glistening orange yellow flakes" 2. Represent fragmented emboli at arteriole bifurcations 4. Neck Exam 1. Carotid Bruit IX. Exam 1. See Eye Vital

-Retinal membrane 4. Retinal Artery Occlusion 1. Retina appears pale-gray due to Retinal edema 2. Central Macula (fovea) with cherry-red spot on white-yellow background 3. Constricted arterioles 4. Box-Carring of Retinal vessels 1. Retinal vessels with interrupted columns of blood appear as train box cars 5. Hollenhorst Plaques (emboli) 1. "Glistening orange

a pale, opaque fundus with a red fovea (cherry-red spot). Typically, the arteries are attenuated and may even appear bloodless. An embolus (eg, a cholesterol embolus, called a Hollenhorst plaque) is sometimes visible. If a major branch is occluded rather than the entire artery, fundus abnormalities and vision loss are limited to that sector of the retina.Patients who have giant cell arteritis are 55

atheroembolism rarely causes hypertension. Abdominal pain, nausea, and vomiting can result from concomitant compromised arterial microcirculation of abdominal organs (eg, pancreas, gastrointestinal tract). Sudden blindness and formation of bright yellow retinal plaques (Hollenhorst plaques) can result from emboli in retinal arterioles.Signs of widespread peripheral embolism (eg, livedo reticularis, painful