Systemic lupus erythematosus mimicking retinalmigraine: a case report. Retinalmigraine is a diagnosis of exclusion and is characterized by repeated episodes of transient monocular blindness associated with migraine. We report a case of systemic lupus erythematosus with acute episodes mimicking retinalmigraines. A 46-year-old woman with a history of migraine with aura since her 20s and Evans syndrome presented with episodic transient monocular blindness. Retinalmigraine was considered as the cause, and migraine prophylaxis initially reduced its frequency. After 5 months, the frequency increased, with chilblain-like lupus lesions on her extremities. Laboratory testing revealed lymphopenia and hypocomplementemia, fulfilling the diagnostic criteria for systemic lupus erythematosus, which may
Migrainous infarction of the eye: Two cases of monocular ischemic complications associated with retinalmigraine. Retinalmigraine is defined by fully reversible monocular visual phenomena. We present two cases that were complicated by permanent monocular vision deficits. A 57-year-old man with history of retinalmigraine experienced persistent monocular vision loss after one stereotypical retinalmigraine, progressing to finger-count vision over 4 days. He developed paracentral acute middle maculopathy that progressed to central retinal artery occlusion. A 27-year-old man with history of retinalmigraine presented with persistent right eye superotemporal scotoma after a retinalmigraine. Relative afferent pupillary defect and superotemporal visual field defect were noted, consistent
Case Report: New-Onset RetinalMigraine After Transseptal Catheterization. While new-onset migraine headaches and binocular visual aura have been reported after transseptal catheterization (TSC), this case suggests that retinal aura may emerge also after this procedure. This 38-year-old male with paroxysmal atrial fibrillation had received TSC and cryoablation, and subsequently developed additional, stereotyped episodes (duration = 20-30 minutes) with zigzag lines and flickering small bright dots in the central visual field of one eye (moving laterally) occurred. A central scotoma was noted during one episode. This is the first case with retinal aura phenomena meeting International Classification of Headache Disorders diagnostic criteria for retinalmigraine, suggesting that this rare
A case report of isolated orbital vasculitis mimicking retinalmigraine: A potential cause of recurrent transient monocular blindness and ipsilateral headache. Retinalmigraine is an important differential diagnosis of recurrent transient monocular blindness accompanied by headache when other etiologies are excluded. Here, we report a case of orbital vasculitis which initially mimicked retinalmigraine. A 47-year-old woman had recurrent episodes of fully reversible transient monocular blindness accompanied by ipsilateral headache for 15 months. The patient's neuroimaging and cardiac and ophthalmologic evaluations were normal. With a diagnosis of retinalmigraine, her symptoms remitted in response to prophylactic treatment with topiramate and propranolol for 8 months. Three months after
presenting with TMVL be referred for neuroimaging and cardiac workup so that appropriate treatments can be initiated to prevent future complications. Headaches during the vision loss may not always indicate a benign cause, and retinalmigraine should be a diagnosis of exclusion.
* Idiopathic intracranial hypertension! * Retinalmigraine * Rare syndrome consisting of recurrent attacks of monocular visual dysfunction, including positive features (such as, scintillations) or negative features (such as, blindness). As with aura, these symptoms are completely reversible. * Hemiplegic migraine * Motor aura consisting of hemiparesis or hemiplegia. The progression of the motor deficit 1. 1. Retinalmigraine 2. Hemiplegic migraine 3. Brainstem migraine 4. Status migrainosus! See above for information!3) What is the pathophysiology of idiopathic intracranial hypertension? Describe the typical patient. * Young, obese, women of childbearing age * CSF prod/absorp. Imbalance. Poorly understood “PATHOGENESIS — Although many theories for IIH have been proposed
pressure trauma or optic nerve drusen.Rarely, retinalmigraine (vasospasm) - this can be seen in younger individuals where all other causes have been excluded.[4]Retinal damage becomes increasingly irreversible over time. The optimal treatment window during which treatment may be beneficial is controversial but believed to be about 90-100 minutes (some say up to 105 minutes)[5] , although some form .2009.01.015. Epub 2009 Apr 18.Chang YS, Ho CH, Chu CC, et al; Risk of retinal artery occlusion in patients with diabetes mellitus: A retrospective large-scale cohort study. PLoS One. 2018 Aug 913(8):e0201627. doi: 10.1371/journal.pone.0201627. eCollection 2018.Grosberg BM, Solomon S, Friedman DI, et al; Retinalmigraine reappraised. Cephalalgia. 2006 Nov26(11):1275-86.Fraser SG, Adams W; Interventions
disturbance, and vertigo.Unilateral symptoms are rare but may occur in retinal or ocular migraine. Retinalmigraine is presumed to result from retinal vasospasm causing isolated hypoperfusion of the retina or the optic nerve. [17, 18] This condition is defined as a fully reversible monocular visual disturbance associated with migraine headache and a normal neuro-ophthalmic examination between attacks . Retinalmigraine is not as common as was once thought; one literature review found only 5 cases meeting the IHS criteria for diagnosis. [19] Intravitreal injection of ocriplasmin, used to treat symptomatic vitreomacular adhesion, has been reported to cause transient vision loss due to presumed direct retinal side effects but with unknown molecular mechanism. [20] Vision loss has also been reported after
to thromboembolic vascular disease, hypercoagulable testing, retinalmigraine, and bilateral vision loss. The objective is to provide the ophthalmologist with information on how to best manage these patients. Thromboembolic etiologies for transient vision loss are sometimes managed with medications, but when carotid surgery is indicated, earlier intervention may prevent future stroke. This need for early . For nonthromboembolic causes of transient vision loss such as retinalmigraine or retinal vasospasm, the ophthalmologist can provide reassurance as well as potentially give medications to decrease the frequency of vision loss episodes.
of the anatomy of visual pathways, clinical features, implications of the visual system on therapy, migraine on visually impaired populations, treatment of visual auras and ocular (retinal) migraine, effect of prophylactic migraine treatments on visual aura, visual symptoms induced by anti-migraine or anti-headache drugs, and differential diagnosis. A comprehensive narrative review from both basic and clinical
include the following: * * Childhood periodic syndromes * * Late-life migrainous accompaniments * * Migraine with brainstem aura * * Hemiplegic migraine * * Status migrainosus * * Retinalmigraine A migraine variant may be suggested by focal neurologic findings, such as the following, that occur with the headache and persist temporarily after the pain
include the following: * * Childhood periodic syndromes * * Late-life migrainous accompaniments * * Migraine with brainstem aura * * Hemiplegic migraine * * Status migrainosus * * Retinalmigraine A migraine variant may be suggested by focal neurologic findings, such as the following, that occur with the headache and persist temporarily after the pain
include the following: * * Childhood periodic syndromes * * Late-life migrainous accompaniments * * Migraine with brainstem aura * * Hemiplegic migraine * * Status migrainosus * * Retinalmigraine A migraine variant may be suggested by focal neurologic findings, such as the following, that occur with the headache and persist temporarily after the pain
Migraine * Episodic Syndromes That May Be Associated with Migraine * RetinalMigraine * Complicated Migraine * Less Common Migraine Variants * Presentation * Differential Diagnosis * CT, MRI, and SPECT * Electroencephalography * Genetic Testing * Treatment * Guidelines * Questions & Answers * * Show All * ReferencesOverview OverviewMigraine is a disorder affecting more than 13% of the general population ), including the following: [2] * * Hemiplegic migraine * * Retinalmigraine * * Chronic migraine * * Episodic syndromes that may be associated with migraine Next: PathophysiologyMigraine is associated with a neuronal network excitability, with activation and sensitization of the trigeminovascular system. Cortical spreading depression (CSD), recognized as the neuronal
include the following: * * Childhood periodic syndromes * * Late-life migrainous accompaniments * * Migraine with brainstem aura * * Hemiplegic migraine * * Status migrainosus * * Retinalmigraine A migraine variant may be suggested by focal neurologic findings, such as the following, that occur with the headache and persist temporarily after the pain
include the following: * * Childhood periodic syndromes * * Late-life migrainous accompaniments * * Migraine with brainstem aura * * Hemiplegic migraine * * Status migrainosus * * Retinalmigraine A migraine variant may be suggested by focal neurologic findings, such as the following, that occur with the headache and persist temporarily after the pain